4Ī prospective diary study quantified migraine aura: Researchers have attempted to study what type of aura people suffer, and it’s plain to see why visual aura is the most commonly thought of, but this also demonstrates how varied aura can be. 2 A second study found that migraine aura with or without headche accounted for 38% of all migraine cases 3
In several large population based studies in Denmark researchers found 32% of people who suffered migraine always suffered migraine with aura, while another 8% suffered both migraine with and without aura. Treat the underlying irritation before attacks begin and deal with a vital piece in the migraine puzzle. It is likely the reason why this then provides successful treatment of aura is due to the LC resuming ‘normal function’ rather than remain irritated. The ability of the Watson Headache ® Approach to decrease underlying ‘noise’ in the trigemino-cervical complex has been validated. Neuroimaging confirms the relationship between the trigemino-cervical complex and the LC changes in the 24 hours prior to an attack. Physiological studies demonstrate the trigemino-cervical complex can cause inhibition of the LC, lowering our stress tolerance, and increasing the likelihood of CSD (aura).
What then, is the ongoing internal source of overload making the system so susceptible to other sources of physiological stress (lack of food, lack of or poor quality sleep, physical stress (over exertion/exercise), certain hormonal events, and injury) to trigger attacks? In keeping with the notion of CSD as one manifestation of the stress response, loss of LC signalling lower the threshold for CSD – i.e. The master of which is the Locus Coeruleus (LC). Migraine can be viewed as the symptomatic expression of an overstimulated stress response system in the brainstem. The diagram depicts the progression of his fortification spectra.ĭespite the presence of an aura, the underlying characteristics of this version of migraine are the same. The location and speed of the wave approximate the quite spectacular images captured here by Hubert Airy in 1870, and repeated several times after. Later researchers examined the rate of spread of Leão’s wave of ‘cortical spreading depression’ (CSD) and reckoned it to be 3mm per minute. Neuronal activity that spread across the surface of the brain. Migraine aura extends to cover a wide range of presentations, with 36% of cases demonstrating paraesthesia – sensory disturbances (pins and needles or numbness) and 10% of cases exhibiting dysphagia (difficulty with speaking), with rarer examples being parosmia (difficulty smelling), dysphagia (difficulty swallowing), dysgeusia (altered taste), hypokinesia (decreased muscle function), paramnesia (deja vu, jamiv vu), auditory hallucinations (tinnitus, buzzing) and altered cognitive function. This is due to the occipital lobe (containing the visual cortex) being the first affected by the changes occurring in the brain during aura.Ĭlassic examples are scintillating scotomas experienced by 50% of people having aura’s – these are the patches of lost or blurred vision that are replaced by geometric shapes (zig-zag lines, crescents, fortification tower shapes, etc). A vast majority (up to 98%) will experience visual disturbances from scotomas or loss of vision and visual snow. So synonymous is aura with migraine, that many people presenting for assessment believe they don’t have migraines ‘because my eyes don’t go funny.’ What is a migraine aura?Īura is defined as a unilateral, fully reversible positive or negative neurological symptom. Migraine aura is one of the most universally thought of symptoms of migraine by the general public, despite affecting less than 30% of migraine sufferers. Oldest description of migraine and aura – Hippocrates 400BC The back part of his head at the vertebrae swelled and the tendons were upon the stretch and hard Not long after, a violent pain seized his right temple, and then his whole head and neck. Flashes like lightning seemed to dart from his eye, and generally his right eye.
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